Percutaneous coronary intervention (PCI) of severe calcified lesions is at high risk
of complications (dissection, perforation, stent underexpansion, etc) and suboptimal
results have been associated with major cardiac adverse events. Intravascular lithotripsy
uses acoustic energy to disrupt the calcified plaque by creating microfractures. The
Shockwave C2 (Shockwave Medical Inc, Santa Clara, CA) intravascular lithotripsy system (S-IVL)
was approved by the Food and Drug Administration after studies confirming its safety
and efficacy.
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References
- Safety and effectiveness of coronary intravascular lithotripsy for treatment of severely calcified coronary stenoses: the Disrupt CAD II Study.Circ Cardiovasc Interv. 2019; 12e008434
- Intravascular lithotripsy for treatment of severely calcified coronary artery disease.J Am Coll Cardiol. 2020; 76: 2635-2646
- Intravascular lithotripsy in calcified coronary lesions: a prospective, observational, multicenter registry.Circ Cardiovasc Interv. 2019; 12e008154
- Coronary lithotripsy for the treatment of underexpanded stents; the international multicentre CRUNCH registry (e-pub ahead of print)..EuroIntervention. 2019; (accessed August 11, 2022.)https://doi.org/10.4244/EIJ-D-21-00545
- IntravaScular Lithotripsy for the Management of UndILatable Coronary StEnt: the SMILE registry.Cardiovasc Revasc Med. 2020; 21: 1555-1559
Article Info
Publication History
Published online: May 25, 2022
Publication stage
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© 2022 Canadian Cardiovascular Society. Published by Elsevier Inc. All rights reserved.
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Access this article on ScienceDirectLinked Article
- Hitting the Rock Behind the Metal: Is Intravascular Lithotripsy for In-Stent Percutaneous Coronary Intervention the Perfect Symphony?Canadian Journal of Cardiology
- PreviewCoronary artery calcification (CAC) adds technical challenge and complexity to percutaneous coronary interventions (PCIs) and is associated with poorer outcomes and major adverse cardiovascular events.1 CAC can lead to stent malapposition, asymmetric expansion, and stent underexpansion, which has been associated with higher rates of device-oriented composite endpoints such as cardiac death, myocardial infarction related to the target vessel, or target-lesion revascularization.
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